Hello Ashmedai, I saw your comments in Igor's substack and appreciate them so I am looking at your substack now. You may have already seen this, but I think the information contained in this conference abstract (there is no underlying paper) from Dr. Stephen Gundry is important as it gives another layer of mechanistic evidence in the myocarditis case study literature. Here is the link;
This was a fairly large population of cardiology patients who had baseline PULS test data prior to getting 2X jabbed. The high level result is that on average the 5-year imputed heart attack risk for the patients increased from 11% --> 25%... a stunning increase. Again, this is based on a series of blood tests and an algorithm used to predict heart attack risk.
Most stunning to me, and the point of my post, is the hepatocypte growth factor (HGF) data that the author broke out. If we assume that the (+/-) ranges are 3-sigma, which is the only thing that makes sense to me if you look at them... then the "before" and "after" population jab data ranges literally have no crossover. This means EVERYBODY was experiencing at least some sub-clinical heart inflammation, signaled by the HGF. Look up HGF.. it is related to antigen driven T-cell infusion into the heart tissue. What could the antigen be? Hmmmmmmmmmmmmmmm
Hello Ashmedai, I saw your comments in Igor's substack and appreciate them so I am looking at your substack now. You may have already seen this, but I think the information contained in this conference abstract (there is no underlying paper) from Dr. Stephen Gundry is important as it gives another layer of mechanistic evidence in the myocarditis case study literature. Here is the link;
https://www.ahajournals.org/doi/abs/10.1161/circ.144.suppl_1.10712?cookieSet=1
This was a fairly large population of cardiology patients who had baseline PULS test data prior to getting 2X jabbed. The high level result is that on average the 5-year imputed heart attack risk for the patients increased from 11% --> 25%... a stunning increase. Again, this is based on a series of blood tests and an algorithm used to predict heart attack risk.
Most stunning to me, and the point of my post, is the hepatocypte growth factor (HGF) data that the author broke out. If we assume that the (+/-) ranges are 3-sigma, which is the only thing that makes sense to me if you look at them... then the "before" and "after" population jab data ranges literally have no crossover. This means EVERYBODY was experiencing at least some sub-clinical heart inflammation, signaled by the HGF. Look up HGF.. it is related to antigen driven T-cell infusion into the heart tissue. What could the antigen be? Hmmmmmmmmmmmmmmm
Thank you for taking time to do this exhaustive research.